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What is Sustanon 250?

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Sustanon® 250 is an oil-based injectable testosterone blend that contains four different testosterone esters: testosterone propionate (30 mg); testosterone phenylpropionate (60 mg); testosterone isocaproate (60 mg); and testosterone decanoate (100 mg). Sustanon® is designed to provide a fast yet extended release of testosterone, usually requiring injections once every 3 to 4 weeks in a clinical setting. This is an improvement from standard testosterones such as cypionate or enanthate, which provide a shorter duration of activity. As with all testosterone products, Sustanon® 250 is a very strong anabolic drug with pronounced androgenic activity. It is most commonly used in bulking cycles, providing exceptional gains in strength and muscle mass.

Administration (Men):
To treat androgen insufficiency, the prescribing guidelines for Sustanon® 250 call for a dosage of 250 mg every 3 weeks. Although active in the body for a longer time, Sustanon® 250 is usually injected every 7 to 10 days for muscle-building purposes. This schedule will allow for the higher doses most commonly applied by athletes, and more stable elevations in hormone level. The usual dosage among male athletes is in the range of 250-750 mg per injection, taken in cycles 6 to 12 weeks in length. This level is sufficient for most users to notice exceptional gains in muscle size and strength.
Sustanon® 250 is usually incorporated into bulking phases of training, when added water retention will be of little consequence, the user more concerned with raw mass than definition. Some do incorporate this drug into cutting cycles as well, but typically in lower doses (125- 250 mg every 7-10 days) and/or when accompanied by an aromatase inhibitor to keep estrogen levels under control. Sustanon® 250 is a very effective anabolic drug, and is often used alone with great benefit. Some, however, find a need to stack it with other anabolic/androgenic steroids for a stronger effect, in which case an additional 200-400 mg per week of boldenone undecylenate, methenolone enanthate, or nandrolone decanoate should provide substantial results with no significant hepatotoxicity. Testosterone is ultimately very versatile, and can be combined with many other anabolic/androgenic steroids to tailor the desired effect.
Some bodybuilders have been known to use excessively high dosages of this drug (1,000 mg per week or more), although this practice is generally not advised. At dosages above 750 mg per week, water retention will likely account for more of the additional weight gain than new muscle tissue. The practice of “megadosing” is inefficient (not to mention potentially dangerous), especially when we take into account the typical high cost of Sustanon 250. Such use is usually not justified outside of aggressive bodybuilding regimens.

Related products:

Sustanon 250 by Aspen

Sustanon by Hilma Biocare

Sustanex by Genexpharma

Llewellyn, William. Anabolics

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Dihydrotestosterone derivatives

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Mesterolone is a potent orally active derivative of dihydrotestosterone. Similar to methenolone, it possesses a non-toxic 1-methyl group, which increases its resistance to hepatic breakdown. This alteration does not increase the stability of the 3-keto group however, and as such, this steroid is a poor anabolic like its parent.
Drostanolone is simply dihydrotestosterone with an added 2-methyl group. This addition greatly increases the stability of the 3-keto group, vital to androgen binding. As such, the activity of this steroid in muscle tissue is greatly enhanced (see: Anabolic/Androgenic Dissociation).
Oxymetholone is an orally active derivative of dihydrotestosterone. The 17-methyl group is well understood at this point as we have discussed it with many steroids, however, the 2-hydroxymethylene group is not seen on any other commercial steroid. We do know that this group greatly enhances anabolic potency by increasing the stability of the 3-keto group, and that the configuration of this substituent also appears to allow this steroid to bind and activate the estrogen receptor.
Stanozolol is a potent anabolic steroid, owing to the fact that the 3-2 pyrazol group creates a stable configuration off the A-ring that allows for androgen receptor binding (this steroid is one of the few that does not possess an actual 3-keto group). As such, it is highly active in muscle tissue, unlike dihydrotestosterone.
Methenolone also is a potent anabolic steroid, due to the fact that the c1-2 double bond increases the stability of the 3-keto group. The 1-methyl group works to increase its oral bioavailability, making methenolone (as methenolone acetate) one of the few orally active non-17-alkylated orals. The c 1-2 bond may also help increase hepatic resistance (slightly) to 17-ketosteroid deactivation as well.
Oxandrolone is an orally active derivative of dihydrotestosterone, due to its 17-methylation. It also differs from DHT by the substitution of its 2-carbon molecule with oxygen. This is the only commercial steroid to carry this group, and further, the only to have a modification to the base carbon structure of the Steran nucleus. The 2-oxo group increases resistance of the 3-keto group to metabolism considerably, making oxandrolone a potent anabolic.

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Nandrolone derivative AAS

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Norethandrolone is simply nandrolone with an added 17-alpha ethyl group. This alteration is rarely used with anabolic/androgenic steroids, and is much more commonly found with synthetic estrogens and progestins. Although 17-ethylation inhibits 17-ketosteroid reduction just as well as 17-methylation, and therefore allows this steroid to exhibit a similarly high level of oral activity, this group also tends to increase progesterone receptor binding. Norethandrolone is clearly a “troublesome” hormone in terms of water retention, fat gain, and gynecomastia, which may in part be due to its heightened binding to this receptor.

Ethylestrenol is an oral derivative of nandrolone, very similar in structure to keto group, which is vital to androgen receptor binding. As such, ethylestrenol is possibly the weakest steroid milligram for milligram ever sold commercially. Any activity this steroid does exhibit is likely from its conversion to norethandrolone, which does seem to occur with some affinity (apparently the 3 oxygen group is metabolically added to this compound without much trouble). This is probably the most interesting trait of ethylestrenol, which is an undistinguished compound otherwise.

Although a derivative of nandrolone, the two additional double-bonds present on trenbolone make any similarities to its parent hormone extremely difficult to see. First, the 9-10 bond inhibits aromatization. Nandrolone is very slowly aromatized, however, some estrogen is still produced from this steroid. Not so with trenbolone. The 11-12 bond additionally increases androgen receptor binding. This steroid also does not undergo 5-alpha reduction like nandrolone, and as such does not share the same dissociation between anabolic and androgenic effects (trenbolone is much more androgenic in comparison).  Click here to see all and their prices.

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Testosterone derivative AAS

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Boldenone is testosterone with an added double-bond between carbon atoms one and two. However, this bond changes the activity of the steroid considerably. First, it dramatically slows aromatization, such that boldenone converts to estradiol at about half the rate of testosterone. Secondly, this bond causes the steroid to be a very poor substrate for the 5-alpha reductase enzyme. The more active 5-alpha reduced metabolite 5alpha-dihydroboldenone is produced only in very small amounts in humans. The hormone instead tends to convert via 5-beta reductase to 5beta-dihydroboldenone (a virtually inactive androgen). This makes it lean towards being an anabolic instead of an androgen, although both traits are still notably apparent with this steroid. The c1-2 double bond also slows the hepatic breakdown of the structure, increasing its resistance to 17-ketosteroid deactivation and its functional half-life and oral bioavailability.

This is the most basic derivative of testosterone, differing only by the added 17- alpha methylation that makes the steroid orally active. Conversion to 17-alpha methylestradiol makes this steroid extremely estrogenic, despite the fact that this alteration actually reduces interaction with the aromatase enzyme.


In many regards, methandrostenolone is very similar to boldenone, as it too exhibits reduced estrogenic and androgenic activity due to the c1-2 double-bond. However, this steroid does have a reputation of being somewhat estrogenic, owing to the fact that it converts to a highly active form of estrogen. Methandrostenolone is also much more active milligram for milligram, as the 17-alpha methyl group also gives it a longer half-life and allows it to exist in a more free state than its cousin boldenone.


Fluoxymesterone is a c-17alpha alkylated oral derivative of testosterone. The 11-beta group functions to inhibit aromatization, so there is no estrogen conversion at all with this steroid. It also works to lower the affinity of this steroid toward restrictive serum binding proteins, increasing its relative activity. Introduction of fluorine at the 9-position also potentiates the action of this steroid.

Llewellyn, William. Anabolics

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Hollywood’s Vial Bodies

Hollywood and Growth Hormone

Hollywood has gone crazy for human growth hormone, with top stars, filmmakers, and studio executives touting its benefits: ripped abs, fewer wrinkles, increased sex drive, more energy (and aggression), etc. With anti-aging clinics prescribing it freely, is H.G.H. a career-saving miracle or a pricey, silly, even hazardous placebo?

It was the moment every movie star fears most. It happened about two years ago, the actor recalls, a tad defensively. There he stood, in all his A-list glory, in front of the full-length mirror in his bathroom. Nine times out of 10, the mirror was just that big shiny thing he brushed past en route from the shower to the bedroom. He wasn’t one of those actors. Despite his natural charisma, or perhaps because of it, he had always scored low on the Celebrity Vanity Index. “Admittedly, it’s a relative scale,” he says. “We do grade on a curve here.”

His first inclination, as he edged closer to his reflection, was to give himself the benefit of the doubt. He’d been around long enough to appreciate the degree to which harsh lighting and bad angles can make anyone look bad. But now, as the steam dissipated and the reflection took on crystal clarity, the time had come to confront a few essential truths. “You know how actors look in movies that ‘age’ their character over several years?” he asks. “I felt like I was glimpsing myself in the Act Three scene that begins with a tagline that reads, ‘Ten Years Later.’ It was kind of a De Niro in Raging Bull moment. Except not award-winning.”

by Ned Zeman

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Fat Loss Agents – Thyroid

What Tiromel is used for

What Tiromel is used for?

Liothyronine sodium is a synthetically manufactured prescription thyroid hormone. It specially consists of the L-isomer of the natural thyroid hormone triiodothyronine (T3). Thyroid hormones stimulate basal metabolic rate, and are involved with many cellular functions including protein, fat, and carbohydrate metabolism. Liothyronine sodium is used medically to treat hypothyroidism, a condition where the thyroid gland does not produce sufficient levels of thyroid hormone. Hypothyroidism is usually diagnosed with a serum hormone profile (T3, T4, & TSH), and may manifest itself with symptoms including loss of energy, lethargy, weight gain, hair loss, and changes in skin texture. T3 is the most active thyroid hormone in the body, and consequently liothyronine sodium is considered to be a more potent thyroid medication than levothyroxine sodium (T4).

Bodybuilders and athletes are attracted to liothyronine sodium for its ability to increase metabolism and support the breakdown of body fat. Most often utilized during contest preparation or periods of “cutting”, the drug is usually said to significantly aid in the loss of fat, often on higher levels of caloric intake than would normally be permissive of such fat loss. To this end, the drug is also commonly used in conjunction with other fat loss agents such as human growth hormone or beta agonists. Some users also ascribe an ability of thyroid hormones like liothyronine sodium to increase the anabolic effect of steroids. While in theory these drugs may support the greater utilization of protein and carbohydrates for muscle growth, they are not widely proven or accepted for this purpose.

When used to treat mild hypothyroidism, the typical recommended starting dosage is 25 mcg daily. The daily dosage then may be increased by no more than 25 mcg every 1 to 2 weeks. The established maintenance dose is usually 25-75 mcg per day. Once a day administration of the full daily dose is usually recommended. Although liothyronine sodium is fast acting, its effects may persist in the body for several days after discontinuance.
The usual protocol among bodybuilders and athletes taking liothyronine sodium to accelerate fat loss involves initiating its use with a dosage of 25 mcg per day. This dosage may be increased by 25 mcg every 4 to 7 days, usually reaching a maximum of no more than 75 mcg per day. As in a medical setting, the intent of this slow buildup is to help the body become adjust to the increasing thyroid hormone levels, and avoid sudden changes that may initiate side effects.
Cycles of liothyronine sodium usually last no longer than 6 weeks, and administration of the drug should not be halted abruptly. Instead, it is discontinued in the same slow manner in which it was initiated. This usually entails reducing the dosage by 25 mcg every 4 to 7 days. This tapering is done so that the body has time to readjust its endogenous hormone production at the conclusion of therapy, and to avoid the onset of side effects.

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Llewellyn, William. Anabolics


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Viagra and Bodybuilding

Viagra and Bodybuilding

Viagra might not be the first supplement you think of when it comes to muscle building. But new science shows this little blue pill might actually be the answer to your bodybuilding prayers.

You’re breathless, sweaty, tired, throbbing and pumped full of blood. You’re fatiguing fast but you know you have to stick with it. Are we on about your final set of a high-volume squat session? Or is this you leaving your girl in a quivering, wet mess on the bed?

Well, it could be either really. Especially if the blue pill of power called Viagra is involved.

Like Arnold said, “I am like getting the feeling of cumming in the gym; I’m getting the feeling of cumming at home; I’m getting the feeling of cumming backstage”.

Who better to say it than the master himself. Maybe he had a pocket full of the stuff too.

So could the medication known as Sidenafil really help build gains?

Get your junk out of your hands bro. Let’s find out…

Viagra is the brand name of sildenafil. It was introduced as a vascular health medication but quickly became a treatment for erectile dysfunction.
As a prescription med, Sidenafil may help to increase muscle function and protein synthesis – but only in untrained men.
Viagra increases testosterone slightly, but not by much.
Side effects of Viagra use include headaches, stomach issues, and blurred vision.

Could Viagra Really Help to Boost Strength and Muscle Mass?
Real bros understand that crafting out a stage-ready physique is more than just a clean diet and throwing a few weights around. You need to grab every inch and tug hard in order to maximize your gains and become the very best in your sport.

Bodybuilding is a lifestyle, a job and a religion that can chew you up and spit you out if you don’t live and breathe it. So when something new comes along that promises to boost muscle mass and strength, it’s worth taking note.

But is sildenafil really as good as it’s claimed to be?

Will it help you take your physique goals to the next level? Or just leave you limp, weak and out of pump?

Viagra enhances blood flow through increased nitric oxide production
In the film The Matrix, taking the blue pill meant the story ended. But it’s the opposite for the little blue Viagra pill, with millions of guys reinvigorating their rock bottom sex lives by slipping it into their nutrition rotation.

What’s interesting to bros around the iron houses of the world though is that Viagra has recently been claimed to offer some potentially exciting performance benefits.

Viagra is classed as a PDE-5 inhibitor. It enhances blood flow by blocking the action of cyclic guanosine monophosphate (cGMP). This helps to allow more blood to your junk, triggers relaxation of smooth muscle, and at the same time stimulate the production of nitric oxide – a blood vessel dilator.

Put all of this together and you’ve got a member with more rock in it than the WWE. This is the main reason it’s used to treat ED – a disorder characterized by a lack of blood flow.
So where does a PDE-5 inhibitor fit into the life of a funk swole brother wanting to gain muscle mass and strength?

Nitric oxide is a compound well known for its ability to enhance and stamina in the gym. In fact, it’s so powerful that nutrients with NO-boosting ingredients are widely used in pre workout supplements to:

Increase power output and strength
Build unbreakable endurance and motivation
Reduce time-to-fatigue

There’s no way Viagra will be as effective as the best nitric oxide-boosting pre workout supplements such as citrulline malate and red beet, but it may still have some value for performance enhancement.

Viagra may increase protein synthesis and reduce fatigue
A recent study from Clinical and Transitional Science made an interesting finding in their study of healthy men taking 25 mg/day sildenafil over a 6-day period.

The men were split into two groups – one was given Viagra and the other a placebo. They both underwent the same tests for fatigue, strength and muscle architecture both before and after treatment.

Hit me bro, what happened?

The research team made some pretty interesting findings. Viagra was able to:

Reduce muscle fatigue
Increase muscle protein synthesis/proteome expression
In theory, these findings could translate to more muscle mass by potentially remodeling muscle tissue and elevating protein levels within muscle cells. Actual changes in muscle growth weren’t measured as such during the study (it was only a short-term project), but the results were interesting.

It’s worth noting though that this study wasn’t done in bodybuilders. It was conducted using older dudes weighing on average 125 lbs who were at risk of muscle loss. It’s not exactly the same as a bunch of bros slamming blue pills to get jacked. These guys were the epitome of ‘DYEL’ers.

On top of that, maximal isometric strength and power didn’t change either.

Does Viagra Increase Testosterone?
As the primary male hormone responsible for ramping up both anabolic and androgenic gains, testosterone is the key player in the war between you and the iron.

Increase your T levels and a bounty of benefits awaits you:

Enhanced muscle mass and strength
Lower body fat and more lean tissue
Increased energy, motivation and determination
Elevated stamina and endurance

That’s to name just a few important, powerful masculinizing benefits.

A study in the aptly named journal Andrology found that when a group of men aged 40-70 with low testosterone was given a high dose of Viagra, their testosterone levels increased.

It wasn’t a huge increase though, with total and free T concentrations increasing by 103 ng.dL and 31.7 pg.mL respectively. That’s nowhere near the increase you’d get from regular strength training or a premium natural testosterone booster. If you want to buy Viagra and similar ED products please click the link here.

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OCT: Off-Cycle Therapy

OCT: Off-Cycle Therapy

The objective of anabolic steroid therapy (when non-medical applications are involved) should be to elicit the desired benefits with the lowest cumulative exposure and side effects. This normally includes diligence with optimizing all aspects of training, rest, and diet, as well as adhering to a Post-Cycle Therapy (PCT) program at the conclusion of each steroid cycle. One the one hand, we want to make each cycle as productive as possible. On the other, we are striving to retain the most gains so the starting point for the next cycle is that much further along. When all aspects are in check, the result should be a need for lower total doses, fewer cycles (longer duration of abstinence), and shorter duration of use on-cycle.
Given the importance of retaining our muscle and performance gains, however, our efforts in this regard should not conclude with Post-Cycle Therapy. Indeed, to receive the greatest long-term benefits from anabolic/androgenic steroid therapy it is also advisable to initiate an Off-Cycle Therapy (OCT) program when the PCT is over. The focus of OCT is typically to use all natural substances (dietary supplements) that favor muscle retention, while simultaneously allowing general physiology and hormonal balances to return. While it is fair and even advisable to approach dietary supplements with a healthy level of skepticism, the field has legitimately advanced enough that we do have products with tangible value. We can find ways to make our programs more effective in the absence of pharmaceuticals.
A well-organized OCT program lasts a minimum of six to eight weeks, and consists of three distinct components. The first is “Testosterone Support,” which seeks to extend an effective PCT program, but with a different and much more basic approach. The second part is “Muscle Cell Re Sensitization.” Heavy training disrupts the muscle cell membranes, so that the muscles become less responsive to exercise stimulation. We want to address this during OCT, and prime the muscles for the next bout of intense training. Lastly, we want to include one or more natural muscle-building substances in the program. This part is called “Anabolic Supplementation”. If the right products are used, distinct anabolic/anti-catabolic effects should be noticed, and more muscle mass will be retained in the long run. All three OCT components are taken simultaneously, sometimes for the full period between the end of PCT and the start of the next AAS cycle.

Part I: Testosterone Support

The testosterone support aspect of our OCT program is substantially different than what is used during traditional PCT. We are no longer looking to aid endogenous testosterone production with anti-estrogenic drugs like tamoxifen or clomiphene, nor to use pharmaceuticals that mimic endogenous luteinizing hormones such as hCG. All pharmaceutical strategies have been concluded at this point, and hopefully have elicited the necessary effects. During OCT, we want to provide our bodies some of the natural components used in the synthesis of testosterone. We want to augment our own natural processes, not artificially shift them.
Vitamin D/Calcium/Zinc
The first thing to pay special attention to during OCT is our vitamin and mineral status, particularly those components that are integral to testosterone biosynthesis. This includes Vitamin D, Calcium, and Zinc. To begin with, clinical studies have shown that higher levels of Vitamin D in the blood are associated with increased testosterone output. Thus, supplementing Vitamin D may be advantageous during the long OCT period, when you will be relying solely on your natural testosterone for the hormonal support of anabolism. Calcium is another nutritive component involved in hormone function, especially the level of bio-available (free) testosterone. A dose of 500-1,000 mg daily may be useful if dietary sources are insufficient. Lastly, a small dose of zinc may also be taken if needed, as this mineral again is tied to androgen biosynthesis. Any deficiency in zinc will likely translate into suppressed (sub-optimal) testosterone output.
D-aspartic Acid
D-aspartic acid (DAA) may also be useful during OCT. DAA is an amino acid that is naturally found in the nervous and endocrine systems, and is believed to play roles in such things as neurotransmission, spermatogenesis, and hormone biosynthesis. Clinical studies that gave 3.2 g/day of D-aspartic acid per day (as sodium salt) to healthy men resulted in a 42% increase in serum testosterone levels in most subjects. This same dose is recommended during OCT.

Part II: Cell Re-sensitization

Repeat high intensity exercise, especially resistance training, causes disruption of the muscle cell membranes. This disruption is in many ways desirable, as it is needed to initiate muscle growth and repair. Without damage, there is no progress. There are some negatives to regular disruption of the muscle cells, however. One of the most fundamental is that the outer membranes of the muscle cells (which consist mainly of fatty acid compounds called phospholipids) are rearranged. In particular, the concentration of arachidonic acid (ARA) is lowered. ARA supports the local anabolic process. Likewise, its depletion is one of the common factors in training stagnation.
Arachidonic Acid To help replenish membrane phospholipids and restore muscle cell responsiveness to training, arachidonic acid should be supplemented during the OCT period. A daily dose of 250 mg is recommended, which represents 50-100% of the normal daily dietary intake of ARA. This amount should be sufficient for phospholipid replenishment, and acceptable for long-term use. Higher doses (500-1,000 mg per day) may provide a more distinct muscle-building effect, but should be limited to six to seven weeks.
Fish Oil
It may also be useful to supplement with fish oil during the OCT period. The main interests are docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA), two Omega-3 essential fatty acids that are also important constituents of muscle cell membrane phospholipids. Additionally, studies suggest that Omega-3 essential fatty acids may enhance the membrane storage of arachidonic acid under some conditions, and thus may indirectly support the pro-anabolic effects of this EFA. A daily dose of 2 grams of fish oil is typically recommended during an Off-Cycle Therapy program.

 Part III: Anabolic Supplementation

An optimal Off-Cycle Therapy program should also include natural products with anabolic/anti-catabolic properties. Many AAS users are skeptical of muscle-building supplements, and rightfully so. The market can be very unreliable, with even the best products falling far short of AAS in terms of efficacy and reliability. Still, the field has progressed a great deal over the years, and there are many products of tangible value. And even a partial muscle sparing effect during the OCT period is highly desirable, as it can significantly alter the baseline muscle level by the start of the next steroid cycle (and thus may influence the timing, dose, or duration of AAS required). It is recommended to limit supplementation to only those ingredients with proven anabolic effects in humans.
Creatine Monohydrate
Creatine monohydrate is regarded as the “original” anabolic supplement, as it was the first to offer substantial performance and body composition improvements for most users. It is typically taken for 8-12 weeks or longer (sometimes throughout the entire OCT period), at a dose of 5 grams per day. Creatine augments muscle size and performance through several distinct mechanisms. The two most prominent are cell volumization (water retention) and cell energy enhancement (cellular ATP re-synthesis), although the supplement also has direct protein synthetic and anti-catabolic properties.
Beta Alanine
Beta Alanine is a non-essential amino acid that serves as a direct precursor for carnosine synthesis. During exercise, hydrogen ions are produced in the muscle cells, which cause the pH level to drop. This precipitates muscle fatigue. Carnosine acts as an intramyocellular buffering agent, countering the build-up of hydrogen ions. By serving as the rate-limiting step in the synthesis of muscle carnosine, beta-alanine is a strong stabilizer of muscle pH.  A dose of 3-6 grams per day is typically used, which should allow the individual to perform measurably longer during training. While this may not be a direct anabolic effect, over time the increased training stimulation can lead to greater muscle preservation/gains.
Branched-Chain Amino Acids
There are three essential Branched Chain Amino Acids (BCAA) – leucine, isoleucine, and valine. These amino acids are very abundant in skeletal muscle protein, accounting for 14-18% of the total content. Supplementation with BCAAs is desirable for a couple of reasons. The first is that they provide integral building blocks for the synthesis of new muscle protein. From a nutritive standpoint, BCAA supplements are very useful. Moreover, BCAA appear to directly stimulate muscle cells to synthesize and retain protein. They are, in fact, among a small selection of clinically validated anabolic supplements in humans. A dosage of 10 grams per day (post-training) is most often used.

Llewellyn, William. Anabolics

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America Loves Steroids

Every sports federation on earth has banned steroid use. In the U.S., there are federal and state laws against their importation, manufacturing, sale, and distribution, with penalties as high as 30 years in prison. Despite all that, the use of anabolic steroids is still on the rise.

This growth in their use isn’t just limited to elite athletes who consider these drugs just as vital to their performance enhancement as protein and omega-3’s, but also among a rather broad swath of our culture. Amateur athletes have embraced them, as well as the regular gym dude who just wants to look good. Aging men covet them to fight off the effects of aging and teens use them to bolster up self esteem, despite all society has done to expose their alleged evils.

Clearly, statistics show that we’re increasingly not just saying “no” to these drugs.

In his 1993 book, “Anabolic Steroids In Sport And Exercise,” Dr. Charles Yesalis, professor of health and human development at Penn State University and a renowned expert on steroids, speculated that 1.2 million American adults purportedly used steroids.

I suspect that the number has mushroomed to somewhere between 12 and 15 million. As evidence, consider how big a role the internet has played with its gigantic network of forums and discussion boards covering every drug.

Look too at the ever-expanding network of domestic underground labs; the proliferation of anti-aging clinics and doctors willing to prescribe testosterone to treat low T in men; the growth in population, the role of social media, and the millions who follow outrageous drug user/celebrities and noted steroid gurus all over cyberspace and you might think my number conservative.

The prevailing attitude regarding the legality of steroids among the bodybuilderatti is of course as blasé as a 70’s rocker lighting up a joint at an AC/DC concert, but familiarity of steroids has gone far beyond that. They’ve become a household word and infiltrated American culture to the degree that the description “on steroids” is now permanently etched in the lexicon. It’s used to soup-up many a mainstream advertising campaign and even to describe fierce tropical storms.

Greg Valentino said it best in the “Bigger Stronger Faster” documentary: “Steroids are as American as apple pie.”

by John Romano at T Nation

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Essentiale Forte (Liver Detoxification)

Essentiale forte N is the trade name for a liver-support supplement distributed in Europe by Aventis Pharma. While this is regarded as a medication in some regions, it actually contains a selection of natural vitamins and phospholipids. Likewise, in many areas, including the United States, Essentiale forte N is sold over the counter. The product specifically contains a complex of B vitamins, vitamin E, and phospholipid forms of linoleic, linolinec, and oleic acid. Essentiale forte N is used widely in Europe to treat cases of hepatic dysfunction, such as those caused by chronic infection, allergy, drug toxicity, or other disease. Essential forte N is of interest to steroid using bodybuilders and athletes for its ability to reduce the level of liver strain caused by anabolic/androgenic steroids (particularly those compounds that are c-17 alpha alkylated).
The main mechanism of action with Essential forte N appears to be focused on the supply of antioxidants and building blocks necessary for the repair of damaged cells. This product contains mainly polyunsaturated phospholipids (mostly phosphatidylcholine), and a complex of B and E vitamins. Phosphatidylcholine is identified as a membrane lipid, and is a key component of the Essential forte N formula. This phospholipid is important to the integrity of cells, adding both flexibility and strength. Phosphatidylcholine has long been identified as an important supplement for the liver, supporting normal hepatic fat metabolism and overall liver health. It is also believed to be an important antioxidant for liver and pancreatic wellness.The additional vitamins in the Essential forte N formula are likely included to increase the antioxidant and regenerative properties of the medication.
Essentiale forte N was the first product shown to mitigate the hepatotoxic effects of anabolic steroid use in a clinical study. The investigation looked at the effects of steroid abuse (with or without Essential forte N), and compared them to controls (non-steroid-using subjects). A full panel of liver enzymes was used to determine the level of relative hepatic strain. As expected, the steroid-only group noticed a significant elevation in liver enzymes that were well above the normal range. Liver enzymes were also elevated in the steroid users taking Essential forte N, however, they were similar to controls and remained within the normal range.The researchers concluded:“The positive association of the abuse severity with the increased hepatic enzymes’ levels suggest a relationship between abused AAS and hepatic cell damage. However, when AAS were taken with [Essentiale forte N], … the hepatotoxic effect appears to be attenuated.” While this one study does not assure that steroid liver toxicity can be completely eliminated, it does lend strong support for the use of Essential forte N with hepatotoxic anabolic steroids.

Llewellyn, William. Anabolics

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